Vol. 9, Issue 1, Part G (2024)

Author(s): Kavita Jaidiya, Chitra Jaidiya, Sunita Kanwar and Sulochana Dariya

Abstract: A 10 cattle’s were presented for treatment in the CVAs, Bikaner with history accidental feeding of grain, chapattis and rice. During clinical examination rectal temperature, ruminal motitlity, respiratory rate, Heart rate were taken. Ruminal fluid collected for Macroscopic, Microscopic and Biochemical examination for diagnosis of disease. There was a significant decrease in mean PH of rumen fluid in all the cattle and falls to 4.7+0.29 in severe acidosis. Smell of ruminal fluid was sour due to rise in concentration of lactic acid. Colour of Rumen liquor in the study was light grey to grayish in colour. acidosis Consistency of rumen liquor was slight viscous. Motility of rumen protozoa was slow. It might be due to low intracellular and environmental PH and high toxicity of rumen encountered in lactic acidic animal. All cattle treated with Magnesium hydroxide, 400gm dissolved in 10 liter water was pumped into rumen by stomach tube to neutralize the acidity. Magnesium hydroxide is a potent alkalizing agent to be used in ruminants as an antacids (Smith and Correa, 2004). This was followed by fluid therapy in the form of Nacl solution 1000ml as I/V drip to counter dehydration and electrolyte imbalance. An injection of thiamin was also given. Soduim bicarbonate, 400gm was advised to be orally drenched twice daily for two days (Suzuki et al. 1999). Alkalizing drugs such as calcium carbonate, magnesium carbonate, and sodium bicarbonate were administered orally. In addition to antibiotics, benzyl penicillin and oxytetracyclin were employed to eradicate gram-positive germs. B-complex with anti-histaminic preparation and liver extract cud transplantations, diets, and supportive care were utilized to restore the ruminal microflora. After two days of treatment, the cow began to normal feed intake and was clinically sound.